Two call for papers for Special Issues on the neurobiology of grief and polycystic ovary syndrome
Journal of Neuroendocrinology is excited to announce two new call for papers for Special Issues on the neurobiology of grief and polycystic ovary syndrome (PCOS).
Submit papers via the Journal website: onlinelibrary.wiley.com/page/journal/13652826/homepage/call-for-papers
Neurobiology of Grief International Network – Europe!
Submission deadline: Tuesday, 31 May 2026
Whether the loss of a loved one or other severe blows of fate – grief affects everyone at some point in their lives. But what exactly happens in the brain and the body during this process? How do we adapt to the experience of loss over time? Why do some people experience particularly long or intense grief, and what are the resilience factors to help others recover more quickly? How can we better support people during difficult periods of grief? These are the questions being explored by researchers in the Neurobiology of Grief International Network (NOGIN), who come from a wide range of disciplines – from basic research to immunology and psychology.
In September 2025, NOGIN was held in Regensburg, Germany, where experts in the field shared their latest advances in the underlying neurobiological basis of grief in humans and animal models alike.
To coincide with this meeting, the Journal of Neuroendocrinology is calling for submissions of original studies, review articles, and commentaries relating to any aspect of the neurobiology of grief within the journal's remit.
Topics of interest for this call for papers include but are not restricted to:
- Basic research on social loss
- Clinical findings
- Clinical applications
- Mood disorders
- Neuroendocrine mechanisms
- Neuropeptides
- Genetics
- Neurophysiology
- Measurement of grief
- Translational studies of grief
Lead Guest Editor:
Oliver Bosch
University of Regensburg
Germany
Co-Guest Editors:
Dr. Beate Ditzen
University of Zurich
Switzerland
Jonathan Singer
Texas Tech University
United States
Neuroendocrine contributions to polycystic ovary syndrome (PCOS)
Submission deadline: Friday, 31 July 2026
Polycystic ovary syndrome (PCOS) is the most common cause of anovulatory infertility, affecting over 100 million individuals globally. The syndrome is characterized by reproductive dysfunction but is also strongly associated with severe comorbidities, including metabolic and cardiovascular diseases, gynecological cancers, adverse outcomes during fertility treatments and pregnancy, and mood disorders. The origin of the syndrome remains unknown. Therefore, there is currently no known cure.
Since the 1970s, PCOS has been associated with an increased frequency of pulsatile luteinizing hormone (LH) release. This has implicated neural circuits that regulate fertility in the pathogenesis of PCOS, recognizing it as a common neuroendocrine disorder. Numerous animal models have been developed to investigate the cause of LH hypersecretion, revealing various changes in neural circuits responsible for reproductive capacity and sex steroid hormone secretion. However, the etiology and progression of alterations to neural circuits, the effects of these changes on PCOS symptoms and comorbidities, and the application of these findings in clinical settings remain unclear.
This special issue will encourage the submission of original and review articles from researchers and clinicians to share emerging ideas on the role of neuroendocrine dysfunction in PCOS. We will also encourage multi-author submissions to integrate hypotheses concerning the etiology, symptomatology, modeling, and treatment of PCOS. Together, this collection aims to provide a comprehensive and updated review that fosters critical thought for future research and treatment.
We invite submissions on any aspect of the melanocortin biology and clinical applications within the remit of the Journal of Neuroendocrinology, including (but not limited to):
- Pathogenesis of PCOS phenotypes;
- Developmental programming of PCOS;
- The identity of central circuits altered in PCOS;
- Impact of neuroendocrine dysfunction on cardiovascular, metabolic and psychiatric morbidities associated with PCOS;
- Novel targets for the treatment and prevention of PCOS;
- Novel methods and approaches to study and treat neuroendocrine dysfunction in PCOS.
Lead Guest Editor:
Dr. Aleisha Moore
Kent State University,
United States
